SHE might be considered one of Hollywood’s sexiest stars, but Salma Hayek hates her body.

The actress - who is married to French billionaire François-Henri Pinault - has revealed how she won a “Best Body Award” - but was too embarrassed to accept it.

“I won a ‘Best Body Award’ from Fitness Magazine and I was too embarrassed to accept it,” Salma said. “I actually don’t have a good body, but if everybody thinks so, I guess it means I’m a good actress.

“I have acted the part of the girl who has a very good body. If you know how to dress, there’s some tricks you can pull.”

Salma, 43, also revealed what she’d be doing if she didn’t make a success of her acting career.

“Growing up, I thought about wanting to be a contortionist or maybe a trapeze artist,” she said. “I would have loved to do something gymnastic. I had another fantasy. Have you ever heard of the group ‘Up With People?’ They’re not a circus troupe, but they came to our city in Mexico when I was a kid doing motivational performances and singing songs about the power we have to change things.

“I had a dream of going away with them - just going from town to town and being in their show to help promote world peace. That was my real secret fantasy.”

Hayek - who got wed earlier this year - recently revealed that she’s still struggling to adjust to married life, especially her husband’s traditions.

“Eating oysters for Christmas is a weird one I didn’t know about,” she said. “I had no idea that would be happening. I’m used to turkey. It takes some getting used to.”

SEOUL, South Korea -

The Internet is set to undergo one of the biggest changes in its four-decade history with the expected approval this week of international domain names - or addresses - that can be written in languages other than English, an official said Monday.

The Internet Corporation for Assigned Names and Numbers, or ICANN - the non-profit group that oversees domain names - is holding a meeting this week in Seoul. Domain names are the monikers behind every Web site, e-mail address and Twitter post, such as “.com” and other suffixes.

One of the key issues to be taken up by ICANN’s board at this week’s gathering is whether to allow for the first time entire Internet addresses to be in scripts that are not based on Latin letters. That could potentially open up the Web to more people around the world as addresses could be in characters as diverse as Arabic, Korean, Japanese, Greek, Hindi and Cyrillic - in which Russian is written.

“This is the biggest change technically to the Internet since it was invented 40 years ago,” Peter Dengate Thrush, chairman of the ICANN board, told reporters, calling it a “fantastically complicated technical feature.” He said he expects the board to grant approval on Friday, the conference’s final day.

The Internet’s roots are traced to experiments at a U.S. university in 1969 but it wasn’t until the early 1990s that its use began expanding beyond academia and research institutions to the public.

Rod Beckstrom, ICANN’s new president and CEO, said that if the change is approved, ICANN would begin accepting applications for non-English domain names and that the first entries into the system would likely come sometime in mid 2010.

Enabling the change, Thrush said, is the creation of a translation system that allows multiple scripts to be converted to the right address.

“We’re confident that it works because we’ve been testing it now for a couple of years,” he said. “And so we’re really ready to start rolling it out.”

Of the 1.6 billion Internet users worldwide, Beckstrom - a former chief of U.S. cybersecurity - said that more than half use languages that have scripts based on alphabets other than Latin.

“So this change is very much necessary for not only half the world’s Internet users today, but more than half of probably the future users as the use of the Internet continues to spread,” he said.

Beckstrom, in earlier remarks to conference participants, recalled that many people had said just three to five years ago that using non-Latin scripts for domain names would be impossible to achieve.

“But you the community and the policy groups and staff and board have worked through them, which is absolutely incredible,” he said.

ICANN is headquartered in the United States in Marina del Rey, California.

NEW YORK - They dashed from the dugout and in from the outfield, swarming Alex Rodriguez in a sea of pinstripes only steps from his spot at third base. “I couldn’t be more excited,” he said. “I feel like a 10-year-old kid.” Making it to the World Series for the first time after all those misses will do that to you.

The New York Yankees, baseball’s biggest spenders, finally cashed in with their first pennant in six years Sunday night, beating the Los Angeles Angels 5-2 in Game 6 of the AL championship series behind the savvy pitching of that old October pro, Andy Pettitte.

Next up, New York hosts defending champion Philadelphia in the World Series opener Wednesday night. Cliff Lee is expected to face ALCS MVP CC Sabathia in an enticing pitching matchup between former Cleveland teammates - and the past two AL Cy Young Award winners.

Ridiculed in the past for his October flops, the three-time MVP played a huge role in helping his team advance through the playoffs, batting .438 with five home runs and 12 RBIs. Thriving under late-inning pressure this time around, the slugger earned his first trip to the Fall Classic during a 16-year career in which he’s accomplished almost everything else.

“That’s what you play for,” Rodriguez said. “In order to win a World Series, you have to get there first.”

Cameras flashed in the stands throughout the ninth inning as the crowd roared louder and louder with each pitch.

After Mariano Rivera fanned pinch-hitter Gary Matthews Jr. for the final out at 1 minute past midnight, Mark Teixeira, Derek Jeter and most Yankees rushed to mob a jubilant Rodriguez near third base.

Rivera received a huge hug from catcher Jorge Posada in front of the mound. Then, Rodriguez and the Yankees partied with beer and bubbly in their swanky, high-tech clubhouse.

“I feared that I wouldn’t be able to contribute, so I had a lot of limitations,” Rodriguez said about his previous playoff failures. “The whole year for me was about trusting my teammates and being one of the guys.”

Pettitte set a postseason record for wins, Johnny Damon hit a two-run single and Rivera closed it out in familiar fashion with a six-out save as the $201 million Yankees won their 40th American League crown by vanquishing the Angels, a longtime nemesis.

“It’s really not a surprise that we are here. I hate to sound like that,” said Sabathia, signed along with fellow free agents Teixeira and A.J. Burnett in a $423.5 million offseason spending spree.

Now, the Yankees go for their record 27th title - when manager Joe Girardi was hired two years ago, he took jersey No. 27 with that in mind.

Not a bad way for Jeter, Posada and crew to finish up the first season at the team’s new $1.5 billion ballpark. As Yankee Stadium grew dark, Sabathia’s and Girardi’s kids ran around the bases on an empty infield.

“We want to enjoy this tonight. We’ll worry about Philly tomorrow,” Jeter said. “Hopefully, we can play one more great series.”

For manager Mike Scioscia and his sloppy Angels, it was their latest playoff failure during a decade of steady regular-season success. Since winning their only championship in 2002, the Angels are yet to return to the World Series despite five AL West titles in the past six years.

“At times we played good baseball. At times we shot ourselves in the foot,” Scioscia said.

After rain postponed Game 6 for a day, the clear weather and mild, 58-degree temperature at first pitch was a stark change from the first two games of the series, when the Angels froze up in the raw chill at Yankee Stadium.

Pettitte escaped a jam in the sixth, going to 3-0 on Kendry Morales before knocking down a comebacker with runners at second and third to preserve a 3-1 edge. The left-hander pumped his fist, then headed for the dugout.

With one on and one out in the seventh, Pettitte left to a standing ovation and tipped his cap to the sellout crowd of 50,173, the largest at the new ballpark. He earned his 16th postseason win, breaking a tie with John Smoltz, and his fifth to close out a postseason series - also a major league record.

“We’ve got a lot of confidence in Andy when he’s on the mound. He’s been a big-game pitcher for us for 14 years,” Jeter said.

Joba Chamberlain got two key outs and Girardi went to a well-rested Rivera in the eighth. He gave up a two-out RBI single to Vladimir Guerrero, making it 3-2, then retired Morales to end the inning.

A diving play by Teixeira at first base helped Rivera avoid further damage.

It was the first earned run allowed at home by the 39-year-old Rivera in a postseason save situation. But the Yankees added two insurance runs in the eighth on a pair of Angels errors and Teixeira’s sacrifice fly.

Rivera finished up for his record 37th postseason save, and the Yankees had their elusive pennant.

“You bring him in, you feel like the game’s over. He’s the best,” Pettitte said.

Rodriguez reached base all five times up and drew a bases-loaded walk in the fourth that put New York ahead 3-1. Earlier in the inning, Damon gave the Yankees the lead with a single off 16-game winner Joe Saunders.

Including their unprecedented collapse against Boston in 2004, the Yankees had lost five straight times with a chance to close out an ALCS - and six in a row with an opportunity to end a playoff series.

But this time, New York got it done with leadership from Jeter, Pettitte, Rivera and Posada, all part of the late 1990s dynasty under manager Joe Torre.

As for Rodriguez, his tumultuous year began with a tense news conference to admit steroids use from 2001-03 with Texas, then hip surgery that sidelined him until May.

It will end in the World Series.

“Pretty incredible, especially with all the stuff I’ve been through this year,” he said. “I just felt very happy and very blessed, and all I cared about this year was winning games.”

Normally airtight on defense and fundamentals, the Angels made eight errors in the series and several other uncharacteristic mistakes. The miscues continued early in the clincher, when Saunders walked five in 3 1-3 shaky innings and Guerrero was doubled off first base on a shallow fly.

With no Rally Monkey bouncing around the video board in the Bronx, Los Angeles failed to pull off one of its signature comebacks. The Angels trailed in all eight of their playoff victories against New York, including a stirring 7-6 triumph in Game 5 on Thursday night that extended the series.

Looking to lock up the pennant, the Yankees turned to a familiar source of success in Pettitte. The 37-year-old left-hander delivered, allowing only one run for his second closeout win of these playoffs. He also beat Minnesota to complete a first-round sweep.

Always a picture of poise and focus in October, narrowed eyes peering between his cap and glove as he takes his signs on the mound, Pettitte also owns postseason records with 38 starts and 237 1-3 innings pitched.

Pettitte was pitching at home for the Yankees in the postseason for the first time since their last World Series game, a 2-0 loss to Josh Beckett and the Florida Marlins in 2003.

This one was a different story.

“They beat us fair and square,” Angels outfielder Torii Hunter said. “It’s just frustrating right now.”

NOTES: The Phillies won two of three at Yankee Stadium in May. … Rodriguez has hit in 11 straight postseason games. … The Yankees are 5-0 at home this postseason.

WASHINGTON - President Barack Obama declared the swine flu outbreak a national emergency, giving his health chief the power to let hospitals move emergency rooms offsite to speed treatment and protect noninfected patients.

The declaration, signed Friday night and announced Saturday, comes with the disease more prevalent than ever in the country and production delays undercutting the government’s initial, optimistic estimates that as many as 120 million doses of the vaccine could be available by mid-October.

Health authorities say more than 1,000 people in the United States, including almost 100 children, have died from the strain of flu known as H1N1, and 46 states have widespread flu activity. So far only 11 million doses have gone out to health departments, doctor’s offices and other providers, according to the Centers for Disease Control and Prevention officials.

Administration officials said the declaration was a pre-emptive move designed to make decisions easier when they need to be made. Officials said the move was not in response to any single development.

Health and Human Services Secretary Kathleen Sebelius now has authority to bypass federal rules when opening alternative care sites, such as offsite hospital centers at schools or community centers if hospitals seek permission.

Some hospitals have opened drive-thrus and drive-up tent clinics to screen and treat swine flu patients. The idea is to keep infectious people out of regular emergency rooms and away from other sick patients.

Hospitals could modify patient rules - for example, requiring them to give less information during a hectic time - to quicken access to treatment, with government approval, under the declaration.

It also addresses a financial question for hospitals - reimbursement for treating people at sites not typically approved. For instance, federal rules do not allow hospitals to put up treatment tents more than 250 yards away from the doors; if the tents are 300 yards or more away, typically federal dollars won’t go to pay for treatment.

Administration officials said those rules might not make sense while fighting the swine flu, especially if the best piece of pavement is in the middle of a parking lot and some medical centers already are putting in place parts of their emergency plans.

“I think the term emergency declaration sounds more dramatic than it really is,” said Dr. Peter Hotez, a research professor and chairman of the Department of Microbiology, Immunology and Tropical Medicine at George Washington University. “It’s largely an administrative move that’s more preemptive …” He said such a step would give emergency rooms and hospitals the flexibility they need.

The national emergency declaration was the second of two steps needed to give Sebelius extraordinary powers during a crisis.

On April 26, the administration declared swine flu a public health emergency, allowing the shipment of roughly 12 million doses of flu-fighting medications from a federal stockpile to states in case they eventually needed them. At the time, there were 20 confirmed cases in the U.S. of people recovering easily. There was no vaccine against swine flu, but the CDC had taken the initial step necessary for producing one.

“As a nation, we have prepared at all levels of government, and as individuals and communities, taking unprecedented steps to counter the emerging pandemic,” Obama wrote in Saturday’s declaration.

He said the pandemic keeps evolving, the rates of illness are rising rapidly in many areas and there’s a potential “to overburden health care resources.”

The government now hopes to have about 50 million doses of swine flu vaccine out by mid-November and 150 million in December. The flu virus has to be grown in chicken eggs, and the yield hasn’t been as high as was initially hoped, officials have said.

“Many millions” of Americans have had swine flu so far, according to an estimate that CDC Director Dr. Thomas Frieden gave Friday. The government doesn’t test everyone to confirm swine flu so it doesn’t have an exact count. He also said there have been more than 20,000 hospitalizations.

Swine influenza (also called swine flu, hog flu, pig flu and sometimes, the swine) is an infection by any one of several types of swine influenza virus. Swine influenza virus (SIV) is any strain of the Orthomyxoviridae that is endemic in pigs.[2] As of 2009, the known SIV strains include influenza C and the subtypes of Influenza A virus known as H1N1, H1N2, H3N1, H3N2, and H2N3.

Swine influenza virus is common throughout pig populations worldwide. Transmission of the virus from pigs to humans is not common and does not always lead to human influenza, often resulting only in the production of antibodies in the blood. If transmission does cause human influenza, it is called zoonotic swine flu. People with regular exposure to pigs are at increased risk of swine flu infection. The meat of an infected animal poses no risk of infection when properly cooked.

During the mid-20th century, identification of influenza subtypes became possible, allowing accurate diagnosis of transmission to humans. Since then, only 50 such transmissions have been confirmed. These strains of swine flu rarely pass from human to human. Symptoms of zoonotic swine flu in humans are similar to those of influenza and of influenza-like illness in general, namely chills, fever, sore throat, muscle pains, severe headache, coughing, weakness and general discomfort.

Classification

Of the three genera of influenza viruses that cause human flu, two also cause influenza in pigs, with influenza A being common in pigs and influenza C being rare.[3] Influenza B has not been reported in pigs. Within influenza A and influenza C, the strains found in pigs and humans are largely distinct, although due to reassortment there have been transfers of genes among strains crossing swine, avian, and human species boundaries.
Influenza C

Influenza C viruses infect both humans and pigs, but do not infect birds.[4] Transmission between pigs and humans have occurred in the past.[5] For example, influenza C caused small outbreaks of a mild form of influenza amongst children in Japan[6] and California.[6] Due to its limited host range and the lack of genetic diversity in influenza C, this form of influenza does not cause pandemics in humans.[7]
Influenza A

Swine influenza is known to be caused by influenza A subtypes H1N1,[8] H1N2,[8] H2N3,[9] H3N1,[10] and H3N2.[8] In pigs, three influenza A virus subtypes (H1N1, H1N2, and H3N2) are the most common strains worldwide.[11] In the United States, the H1N1 subtype was exclusively prevalent among swine populations before 1998; however, since late August 1998, H3N2 subtypes have been isolated from pigs. As of 2004, H3N2 virus isolates in US swine and turkey stocks were triple reassortants, containing genes from human (HA, NA, and PB1), swine (NS, NP, and M), and avian (PB2 and PA) lineages.[12]
Surveillance

Although there is no formal national surveillance system in the United States to determine what viruses are circulating in pigs,[13] there is an informal surveillance network in the United States that is part of a world surveillance network.

Veterinary medical pathologist, Tracey McNamara, set up a national disease surveillance system in zoos because the zoos do active disease surveillance and many of the exotic animals housed there have broad susceptibilities. Many species fall below the radar of any federal agencies (including dogs, cats, pet prairie dogs, zoo animals, and urban wildlife), even though they may be important in the early detection of human disease outbreaks.[14] [15]
History

Swine influenza was first proposed to be a disease related to human influenza during the 1918 flu pandemic, when pigs became sick at the same time as humans.[16] The first identification of an influenza virus as a cause of disease in pigs occurred about ten years later, in 1930.[17] For the following 60 years, swine influenza strains were almost exclusively H1N1. Then, between 1997 and 2002, new strains of three different subtypes and five different genotypes emerged as causes of influenza among pigs in North America. In 1997-1998, H3N2 strains emerged. These strains, which include genes derived by reassortment from human, swine and avian viruses, have become a major cause of swine influenza in North America. Reassortment between H1N1 and H3N2 produced H1N2. In 1999 in Canada, a strain of H4N6 crossed the species barrier from birds to pigs, but was contained on a single farm.[17]

The H1N1 form of swine flu is one of the descendants of the strain that caused the 1918 flu pandemic.[18][19] As well as persisting in pigs, the descendants of the 1918 virus have also circulated in humans through the 20th century, contributing to the normal seasonal epidemics of influenza.[19] However, direct transmission from pigs to humans is rare, with only 12 cases in the U.S. since 2005.[20] Nevertheless, the retention of influenza strains in pigs after these strains have disappeared from the human population might make pigs a reservoir where influenza viruses could persist, later emerging to reinfect humans once human immunity to these strains has waned.

Swine flu has been reported numerous times as a zoonosis in humans, usually with limited distribution, rarely with a widespread distribution. Outbreaks in swine are common and cause significant economic losses in industry, primarily by causing stunting and extended time to market. For example, this disease costs the British meat industry about £65 million every year.[22]
1918 pandemic in humans

The 1918 flu pandemic in humans was associated with H1N1 and influenza appearing in pigs;[19] this may reflect a zoonosis either from swine to humans, or from humans to swine. Although it is not certain in which direction the virus was transferred, some evidence suggests that, in this case, pigs caught the disease from humans.[16] For instance, swine influenza was only noted as a new disease of pigs in 1918, after the first large outbreaks of influenza amongst people.[16] Although a recent phylogenetic analysis of more recent strains of influenza in humans, birds, and swine suggests that the 1918 outbreak in humans followed a reassortment event within a mammal,[23] the exact origin of the 1918 strain remains elusive. It is estimated that anywhere from 50 to 100 million people were killed worldwide.
1976 U.S. outbreak
Main article: 1976 swine flu outbreak

On February 5, 1976, in the United States an army recruit at Fort Dix said he felt tired and weak. He died the next day and four of his fellow soldiers were later hospitalized. Two weeks after his death, health officials announced that the cause of death was a new strain of swine flu. The strain, a variant of H1N1, is known as A/New Jersey/1976 (H1N1). It was detected only from January 19 to February 9 and did not spread beyond Fort Dix.[26]
President Ford receives swine flu vaccination

This new strain appeared to be closely related to the strain involved in the 1918 flu pandemic. Moreover, the ensuing increased surveillance uncovered another strain in circulation in the U.S.: A/Victoria/75 (H3N2) spread simultaneously, also caused illness, and persisted until March.[26] Alarmed public-health officials decided action must be taken to head off another major pandemic, and urged President Gerald Ford that every person in the U.S. be vaccinated for the disease.

The vaccination program was plagued by delays and public relations problems.[28] On October 1, 1976, immunizations began and three senior citizens died soon after receiving their injections. This resulted in a media outcry that linked these deaths to the immunizations, despite the lack of any proof that the vaccine was the cause. According to science writer Patrick Di Justo, however, by the time the truth was known-that the deaths were not proven to be related to the vaccine-it was too late. “The government had long feared mass panic about swine flu-now they feared mass panic about the swine flu vaccinations.” This became a strong setback to the program.

There were reports of Guillain-Barré syndrome, a paralyzing neuromuscular disorder, affecting some people who had received swine flu immunizations. This syndrome is a rare side-effect of modern influenza vaccines, with an incidence of about one case per million vaccinations.[30] As a result, Di Justo writes that “the public refused to trust a government-operated health program that killed old people and crippled young people.” In total, 48,161,019 Americans, or just over 22% of the population, had been immunized by the time the National Influenza Immunization Program (NIIP) was effectively halted on December 16, 1976.

Overall, there were 1098 cases of Guillain-Barré Syndrome (GBS) recorded nationwide by CDC surveillance, 532 of which were linked to the NIIP vaccination, resulting in death from severe pulmonary complications for 25 people, which, according to Dr. P. Haber, were probably caused by an immunopathological reaction to the 1976 vaccine. Other influenza vaccines have not been linked to GBS, though caution is advised for certain individuals, particularly those with a history of GBS. Still, as observed by a participant in the immunization program, the vaccine killed more Americans than the disease did.[36]
1988 zoonosis

In September 1988, a swine flu virus killed one woman and infected others. 32-year old Barbara Ann Wieners was eight months pregnant when she and her husband, Ed, became ill after visiting the hog barn at a county fair in Walworth County, Wisconsin. Barbara died eight days later, after developing pneumonia.[37] The only pathogen identified was an H1N1 strain of swine influenza virus. Doctors were able to induce labor and deliver a healthy daughter before she died. Her husband recovered from his symptoms.

Influenza-like illness (ILI) was reportedly widespread among the pigs exhibited at the fair. 76% of 25 swine exhibitors aged 9 to 19 tested positive for antibody to SIV, but no serious illnesses were detected among this group. Additional studies suggested between one and three health care personnel who had contact with the patient developed mild influenza-like illnesses with antibody evidence of swine flu infection. However, there was no community outbreak.

In 1998, swine flu was found in pigs in four U.S. states. Within a year, it had spread through pig populations across the United States. Scientists found that this virus had originated in pigs as a recombinant form of flu strains from birds and humans. This outbreak confirmed that pigs can serve as a crucible where novel influenza viruses emerge as a result of the reassortment of genes from different strains.[41][42][43] Genetic components of these 1998 triple-hybrid stains would later form six out of the eight viral gene segment in the 2009 flu outbreak.

On August 20, 2007 Department of Agriculture officers investigated the outbreak (epizootic) of swine flu in Nueva Ecija and Central Luzon, Philippines. The mortality rate is less than 10% for swine flu, unless there are complications like hog cholera. On July 27, 2007, the Philippine National Meat Inspection Service (NMIS) raised a hog cholera “red alert” warning over Metro Manila and 5 regions of Luzon after the disease spread to backyard pig farms in Bulacan and Pampanga, even if these tested negative for the swine flu virus. ]
2009 outbreak in humans
Main article: 2009 flu pandemic

The H1N1 viral strain implicated in the 2009 flu pandemic among humans often is called “swine flu” because initial testing showed many of the genes in the virus were similar to influenza viruses normally occurring in North American swine.[51] Further research has shown that three-quarters or six out of the eight gene segments of the 2009 virus arose from the 1998 North American swine flu strains which emerged from the first-ever reported triple-hybrid virus of 1998.

In late April, Margaret Chan, the World Health Organization’s director-general, declared a “public health emergency of international concern” under the rules of the WHO’s new International Health Regulations when the first cases of the H1N1 virus were reported in the United States.[52][53] Following the outbreak, on May 2, 2009, it was reported in pigs at a farm in Alberta, Canada, with a link to the outbreak in Mexico. The pigs are suspected to have caught this new strain of virus from a farm worker who recently returned from Mexico, then showed symptoms of an influenza-like illness.[54] These are probable cases, pending confirmation by laboratory testing.

The new strain was initially described as an apparent reassortment of at least four strains of influenza A virus subtype H1N1, including one strain endemic in humans, one endemic in birds, and two endemic in swine. Subsequent analysis suggested it was a reassortment of just two strains, both found in swine. Although initial reports identified the new strain as swine influenza (i.e., a zoonosis originating in swine), its genetic origin was only later revealed to have been mostly a descendant of the triple-reassortment virus which emerged in factory farms in the United States in 1998. Several countries took precautionary measures to reduce the chances for a global pandemic of the disease.[56] The 2009 swine flu has been compared to other similar types of influenza virus in terms of mortality: “in the US it appears that for every 1000 people who get infected, about 40 people need admission to hospital and about one person dies.”[57] There are fears that swine flu will become a major global pandemic at the end of the year (coinciding with the Northern Hemisphere winter months), with many countries planning major vaccination campaigns.[58]
Transmission
Transmission between pigs

Influenza is quite common in pigs, with about half of breeding pigs having been exposed to the virus in the US. Antibodies to the virus are also common in pigs in other countries.

The main route of transmission is through direct contact between infected and uninfected animals.[11] These close contacts are particularly common during animal transport. Intensive farming may also increase the risk of transmission, as the pigs are raised in very close proximity to each other.[60][61] The direct transfer of the virus probably occurs either by pigs touching noses, or through dried mucus. Airborne transmission through the aerosols produced by pigs coughing or sneezing are also an important means of infection.[11] The virus usually spreads quickly through a herd, infecting all the pigs within just a few days.[2] Transmission may also occur through wild animals, such as wild boar, which can spread the disease between farms.
Transmission to humans

People who work with poultry and swine, especially people with intense exposures, are at increased risk of zoonotic infection with influenza virus endemic in these animals, and constitute a population of human hosts in which zoonosis and reassortment can co-occur. Vaccination of these workers against influenza and surveillance for new influenza strains among this population may therefore be an important public health measure Transmission of influenza from swine to humans who work with swine was documented in a small surveillance study performed in 2004 at the University of Iowa. This study among others forms the basis of a recommendation that people whose jobs involve handling poultry and swine be the focus of increased public health surveillance. Other professions at particular risk of infection are veterinarians and meat processing workers, although the risk of infection for both of these groups is lower than that of farm workers.
Interaction with avian H5N1 in pigs

Pigs are unusual as they can be infected with influenza strains that usually infect three different species: pigs, birds and humans.[67] This makes pigs a host where influenza viruses might exchange genes, producing new and dangerous strains.[67] Avian influenza virus H3N2 is endemic in pigs in China and has been detected in pigs in Vietnam, increasing fears of the emergence of new variant strains.[68] H3N2 evolved from H2N2 by antigenic shift.[69] In August 2004, researchers in China found H5N1 in pigs.

These H5N1 infections may be quite common: in a survey of 10 apparently healthy pigs housed near poultry farms in West Java, where avian flu had broken out, five of the pig samples contained the H5N1 virus. The Indonesian government has since found similar results in the same region. Additional tests of 150 pigs outside the area were negative.
Signs and symptoms
In swine

In pigs influenza infection produces fever, lethargy, sneezing, coughing, difficulty breathing and decreased appetite. In some cases the infection can cause abortion. Although mortality is usually low (around 1-4%), the virus can produce weight loss and poor growth, causing economic loss to farmers Infected pigs can lose up to 12 pounds of body weight over a 3 to 4 week period.
In humans
Main symptoms of swine flu in humans

Direct transmission of a swine flu virus from pigs to humans is occasionally possible (called zoonotic swine flu). In all, 50 cases are known to have occurred since the first report in medical literature in 1958, which have resulted in a total of six deaths.[74] Of these six people, one was pregnant, one had leukemia, one had Hodgkin disease and two were known to be previously healthy. Despite these apparently low numbers of infections, the true rate of infection may be higher, since most cases only cause a very mild disease, and will probably never be reported or diagnosed.

In this video, Dr. Joe Bresee, with CDC’s Influenza Division, describes the symptoms of swine flu and warning signs to look for that indicate the need for urgent medical attention.
See also: See this video with subtitles on YouTube

According to the Centers for Disease Control and Prevention (CDC), in humans the symptoms of the 2009 “swine flu” H1N1 virus are similar to those of influenza and of influenza-like illness in general. Symptoms include fever, cough, sore throat, body aches, headache, chills and fatigue. The 2009 outbreak has shown an increased percentage of patients reporting diarrhea and vomiting.[75] The 2009 H1N1 virus is not zoonotic swine flu, as it is not transmitted from pigs to humans, but from person to person.

Because these symptoms are not specific to swine flu, a differential diagnosis of probable swine flu requires not only symptoms but also a high likelihood of swine flu due to the person’s recent history. For example, during the 2009 swine flu outbreak in the United States, CDC advised physicians to “consider swine influenza infection in the differential diagnosis of patients with acute febrile respiratory illness who have either been in contact with persons with confirmed swine flu, or who were in one of the five U.S. states that have reported swine flu cases or in Mexico during the 7 days preceding their illness onset.” A diagnosis of confirmed swine flu requires laboratory testing of a respiratory sample (a simple nose and throat swab).

The most common cause of death is respiratory failure. Other causes of death are pneumonia (leading to sepsis)[77], high fever (leading to neurological problems), dehydration (from excessive vomiting and diarrhea) and electrolyte imbalance. Fatalities are more likely in young children and the elderly.
Diagnosis
Thermal scanning of passengers arriving at Singapore Changi airport.
Wiki letter w.svg This section requires expansion.

Different medical kits are available for diagnosis of swine flu.

The two major tests that are being used are the nasopharyngeal (or back of the throat) swab for viral culture, the gold standard, and the indirect evidence test by detection of antibodies to novel H1N1 with PCR studies.
Prevention

Prevention of swine influenza has three components: prevention in swine, prevention of transmission to humans, and prevention of its spread among humans.
In swine

Methods of preventing the spread of influenza among swine include facility management, herd management, and vaccination (ATCvet code: QI09AA03). Because much of the illness and death associated with swine flu involves secondary infection by other pathogens, control strategies that rely on vaccination may be insufficient.

Control of swine influenza by vaccination has become more difficult in recent decades, as the evolution of the virus has resulted in inconsistent responses to traditional vaccines. Standard commercial swine flu vaccines are effective in controlling the infection when the virus strains match enough to have significant cross-protection, and custom (autogenous) vaccines made from the specific viruses isolated are created and used in the more difficult cases. Present vaccination strategies for SIV control and prevention in swine farms typically include the use of one of several bivalent SIV vaccines commercially available in the United States. Of the 97 recent H3N2 isolates examined, only 41 isolates had strong serologic cross-reactions with antiserum to three commercial SIV vaccines. Since the protective ability of influenza vaccines depends primarily on the closeness of the match between the vaccine virus and the epidemic virus, the presence of nonreactive H3N2 SIV variants suggests that current commercial vaccines might not effectively protect pigs from infection with a majority of H3N2 viruses.[81][82] The United States Department of Agriculture researchers say that while pig vaccination keeps pigs from getting sick, it does not block infection or shedding of the virus.

Facility management includes using disinfectants and ambient temperature to control virus in the environment. The virus is unlikely to survive outside living cells for more than two weeks, except in cold (but above freezing) conditions, and it is readily inactivated by disinfectants.[2] Herd management includes not adding pigs carrying influenza to herds that have not been exposed to the virus. The virus survives in healthy carrier pigs for up to 3 months and can be recovered from them between outbreaks. Carrier pigs are usually responsible for the introduction of SIV into previously uninfected herds and countries, so new animals should be quarantined.[59] After an outbreak, as immunity in exposed pigs wanes, new outbreaks of the same strain can occur.
In humans

Swine can be infected by both avian and human influenza strains of influenza, and therefore are hosts where the antigenic shifts can occur that create new influenza strains.

The transmission from swine to human is believed to occur mainly in swine farms where farmers are in close contact with live pigs. Although strains of swine influenza are usually not able to infect humans this may occasionally happen, so farmers and veterinarians are encouraged to use a face mask when dealing with infected animals. The use of vaccines on swine to prevent their infection is a major method of limiting swine to human transmission. Risk factors that may contribute to swine-to-human transmission include smoking and not wearing gloves when working with sick animals.

Prevention of human to human transmission

Influenza spreads between humans through coughing or sneezing and people touching something with the virus on it and then touching their own nose or mouth.[85] Swine flu cannot be spread by pork products, since the virus is not transmitted through food.[85] The swine flu in humans is most contagious during the first five days of the illness although some people, most commonly children, can remain contagious for up to ten days. Diagnosis can be made by sending a specimen, collected during the first five days for analysis.[86]
Thermal imaging camera & screen, photographed in an airport terminal in Greece. Thermal imaging can detect elevated body temperature, one of the signs of the virus N1H1 (Swine influenza).

Recommendations to prevent spread of the virus among humans include using standard infection control against influenza. This includes frequent washing of hands with soap and water or with alcohol-based hand sanitizers, especially after being out in public.[87] Chance of transmission is also reduced by disinfecting household surfaces, which can be done effectively with a diluted chlorine bleach solution.[88]

Experts agree that hand-washing can help prevent viral infections, including ordinary influenza and the swine flu virus. Also avoiding touching eyes, nose and mouth with hands prevents flu.[89] Influenza can spread in coughs or sneezes, but an increasing body of evidence shows small droplets containing the virus can linger on tabletops, telephones and other surfaces and be transferred via the fingers to the mouth, nose or eyes. Alcohol-based gel or foam hand sanitizers work well to destroy viruses and bacteria. Anyone with flu-like symptoms such as a sudden fever, cough or muscle aches should stay away from work or public transportation and should contact a doctor for advice.

Social distancing is another tactic. It means staying away from other people who might be infected and can include avoiding large gatherings, spreading out a little at work, or perhaps staying home and lying low if an infection is spreading in a community. Public health and other responsible authorities have action plans which may request or require social distancing actions depending on the severity of the outbreak.

Vaccination

Vaccines are available for different kinds of swine flu. The Food and Drug Administration (FDA) approved the new swine flu vaccine on September 15, 2009.[91] Studies by the National Institutes of Health (NIH), show that a single dose creates enough antibodies to protect against the virus within about 10 days.
Treatment
In swine

As swine influenza is rarely fatal to pigs, little treatment beyond rest and supportive care is required.[59] Instead veterinary efforts are focused on preventing the spread of the virus throughout the farm, or to other farms.[11] Vaccination and animal management techniques are most important in these efforts. Antibiotics are also used to treat this disease, which although they have no effect against the influenza virus, do help prevent bacterial pneumonia and other secondary infections in influenza-weakened herds.[59]
In humans

If a person becomes sick with swine flu, antiviral drugs can make the illness milder and make the patient feel better faster. They may also prevent serious flu complications. For treatment, antiviral drugs work best if started soon after getting sick (within 2 days of symptoms). Beside antivirals, supportive care at home or in hospital, focuses on controlling fevers, relieving pain and maintaining fluid balance, as well as identifying and treating any secondary infections or other medical problems. The U.S. Centers for Disease Control and Prevention recommends the use of Tamiflu (oseltamivir) or Relenza (zanamivir) for the treatment and/or prevention of infection with swine influenza viruses; however, the majority of people infected with the virus make a full recovery without requiring medical attention or antiviral drugs.[93] The virus isolates in the 2009 outbreak have been found resistant to amantadine and rimantadine.

In the U.S., on April 27, 2009, the Food and Drug Administration (FDA) issued Emergency Use Authorizations to make available Relenza and Tamiflu antiviral drugs to treat the swine influenza virus in cases for which they are currently unapproved. The agency issued these EUAs to allow treatment of patients younger than the current approval allows and to allow the widespread distribution of the drugs, including by non-licensed volunteers.

NEW YORK - Derek Jeter and CC Sabathia got the New York Yankees off to a winning postseason start in their new ballpark. Even Alex Rodriguez broke out of his playoff rut against these tired Minnesota Twins.

Jeter tied it with a two-run homer, Sabathia was the ace the Yankees signed him to be, and New York romped over the Twins 7-2 Wednesday night in the opener of their AL playoff series.

After Jeter’s third-inning homer off loser Brian Duensing pulled New York even at 2-2, Nick Swisher pulled a go-ahead double down the left-field line in the fourth that scored Robinson Cano from first as the Twins made a pair of poor throws.

Rodriguez had gone 0 for 29 in the postseason with runners on base dating to Game 4 of the 2004 AL championship series before chasing Duensing with an RBI single that made it 4-2 in the fifth.

On a night with sustained winds blowing to right-center at 20 mph, with gusts up to 43 mph, Hideki Matsui followed with a two-run homer into Monument Park on left-hander Francisco Lirano’s fourth pitch. The Yankees celebrated like kids, just as the Twins did when they beat Detroit in an AL Central tiebreaker at the Metrodome on Tuesday night. New York went on to win its first playoff game since Joe Girardi replaced Joe Torre as manager after the 2007 season.

Rodriguez added another run-scoring single in the seventh against Jon Rauch following an error by first baseman Michael Cuddyer, with A-Rod’s drive hitting halfway up the right-field wall.

Minnesota didn’t arrive to its hotel until nearly 4 a.m. and appeared to lack the energy that propelled the Twins during a 17-4 finish, which overcame a seven-game division deficit. The teams get a day off before resuming Friday night, when A.J. Burnett pitches for the Yankees against Nick Blackburn. New York will be trying to get off to its first 2-0 postseason start since 1999 against Texas.

Casey Stengel hit the first postseason home run across the street at original Yankee Stadium, an inside-the-parker that gave the New York Giants a 5-4 win in the 1923 World Series opener.

It was Jeter, naturally, who hit the first home run in the Yankees’ $1.5 billion palace, where New York was a major league-best 57-24 at home during the regular season. The captain also got New York’s first hit, was on base four times with two hits and two walks, and scored three runs.

New York won all seven games against the Twins during the season, and was 23-3 at home against Minnesota during the regular season from 2002 on, but the Yankees had split four home games against Minnesota in the playoffs in 2003 and 2004.

After getting past the Twins in 2004 and taking a 3-0 lead against Boston in the ALCS, the Yankees lost 13 of their next 17 postseason games. To rebuild their rotation, the Yankees signed Sabathia and Burnett for a combined $243.5 million.

Sabathia, who had lost his last three playoff decisions for Cleveland and Milwaukee, didn’t disappoint. Wearing long sleeves on the blustery night, he got past a 22-pitch first inning and found a sharp slider. Sabathia allowed one earned run and eight hits in 6 2-3 innings, striking out eight and walking none.

He left with two on after 113 pitches, tipping his hat to a ballpark record crowd of 49,464 that included actress Kate Hudson and rapper Jay-Z. Phil Hughes came in and struck out Orlando Cabrera, one of the Twins’ tiebreaker stars, in a 10-pitch at-bat.

Duensing, who was pitching for the U.S. in the Olympics last year, made just nine starts during his rookie season but was on the mound following the Twins’ busy pennant race run. He gave up five runs and seven hits in 4 2-3 innings.

Sabathia escaped trouble in the first after Denard Span’s leadoff double and the first of Jorge Posada’s two passed balls by striking out AL batting champion Joe Mauer and retiring Cuddyer on a flyout.

Minnesota took a 2-0 lead in the third when Cabrera singled with two outs, Mauer doubled, Cuddyer hit an RBI single and Posada crossed up with Sabathia and allowed a run-scoring passed ball.

Jeter tied the score with a drive about 10 feet fair down the left-field line. With his 18th postseason home run, he tied Yankees stars Mickey Mantle and Reggie Jackson for third on the career list behind Manny Ramirez (28) and Bernie Williams (22), another former New York star.

Swisher doubled in the go-ahead run. Left fielder Delmon Young picked up the ball at the wall and made a one-hop throw to Cabrera, whose one-hop throw up was up the first-base line.

Rodriguez flied out ending the first, extending his playoff hitless streak with runners in scoring position to 19 at-bats, and struck out with a runner on in the third before coming through.

NOTES: Matsui has seven postseason homers. He hit 13 off left-handers during the regular season, tied for big league lead among left-handed batters. … Mariano Rivera pitched the ninth, even though it wasn’t a save situation. He struck out two.